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Description
In research environments, BPC-157 is referenced in preclinical and exploratory studies examining biochemical and molecular mechanisms under controlled laboratory conditions

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Consequently, mitochondrial dysfunction exacerbates oxidative stress, impairs mitochondrial DNA (mtDNA) or deletions of mtDNA, increases intracellular Ca2+ levels, changes in fission/fusion and mitochondrial morphology, and lastly leads to neuronal death

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