glutathione ros mechanism depletion induces oxidative injury and apoptosis via TRPM2 channel activation in renal collecting duct cells ROS Function in Redox Signaling
Description
Additionally, GHK-Cu treatment notably upregulated HO-1 expression (Figure 6G) and downregulated NF-B p65 expression (Figure 6I)
doi: 10.1126/science.abn9257
M., Mullally, J
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