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The release of cytochrome C from the mitochondria leads to production of a caspase-activating complex of cytochrome c, APAF-1, and procaspase-9, triggers the up-regulation of caspase-9, and subsequently increases the expression of caspase-3, followed by the induction of apoptotic events in the cell [44]
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showed that prolactin (PRL) increased the expression and the activity of CPT1A in breast cancer cells with respect to normal cells 29
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45 JaruskovaM.CurikN.HercogR.PolivkovaV.MotlovaE.BenesV.et al (2017)
