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Upon receptor activation, intracellular signalling cascades involving phospholipase C, inositol triphosphate, and calcium mobilisation are initiated, leading to pulsatile release of growth hormone independent of hypothalamic growth hormone releasing hormone (GHRH) stimulation

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Background Cardiac fibrosis following myocardial infarction (MI) drives adverse ventricular remodeling and heart failure, with cardiac fibroblasts (CFs) playing a central role