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Such altered expression by valvular interstitial cells has also been demonstrated to correlate with disease severity

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DOI:10.1093/ajcn/50.2.301

Nature 2015

Given the interesting findings from mechanistic studies in mouse models, where FMO3 suppression reduced TMAO levels and atherosclerosis [32], further research in larger and more standardized human studies is needed to better understand the genetic regulation of TMAO metabolism and its role in CVD risk

In males, the decrease in androgens/AR signaling contributes to the disorder of glucose and lipid metabolism and is considered a promising predictor of chronic diseases