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These effects become therapeutically interesting in obesity because they suggest that glucagon receptor activation could complement appetite suppression by increasing caloric expenditure and improving hepatic lipid handling

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[DOI] [Google Scholar] 205.Spiegel JY et al

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Despite the fact that BSO does not affect overall cellular PDI levels, we found that cells with selective PDI knockdown clearly manifest a markedly-reduced sensitivity to BSO-induced ferroptosis