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ChAT, choline acetyl transferase

Antioxidants are essential for preserving homeostasis and aquatic organisms have been demonstrated to upregulate their antioxidant systems including glutathione peroxidase (GPx), catalase (CAT), and superoxide dismutase (SOD), as well as non-enzymatic free radical scavengers, such as reduced glutathione (GSH) and biotransformation enzymes, such as glutathione-S-transferase (GST)

This excessive production of radical oxidants can occur in pathological states where mitochondrial function is impaired [3], as is common in numerous metabolic diseases [46], as well as infection

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Moving forward, mechanistic in vivo toxicity studies must be prioritized to clarify hazard pathways and establish safety thresholds for BSPs clinical translation