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Abstract Bacterial iron uptake machinery can be hijacked for the targeted delivery of antibiotics into pathogens by attaching antibiotics to siderophores, iron chelators that are employed by bacteria to obtain this essential nutrient

Consequently, systemic oxidative burden may decrease circulating PON1 activity and amplify lipid peroxidation, creating a self-reinforcing cycle that contributes to the pathophysiology of sarcoidosis

Autophagy-endoplasmic reticulum stress inhibition mechanism of superoxide dismutase in the formation of calcium oxalate kidney stones

5662 NAC therapy is recommended in such clinical scenarios, to overcome the reported incidence rates of 60% for severe hepatotoxicity and 5% for mortality

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