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Tumour cells communicate with vascular endothelial cells within developing neoplasms via diffusible growth factors, leading to increased vascularization that further facilitates tumour growth

doi: 10.1111/pme.12081

Conclusion Molecular mechanisms underlying the resistance of pituitary tumor to pharmacological treatment with DA and SS analogs might involve different steps that from the receptor activation by agonist lead to the final biological responses in terms of hormone secretion inhibition as well as tumor shrinkage

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The 2026 ADA Standards of Care and WHO guidelines now recommend GLP-1 therapies for a wide range of metabolic conditions

This framing is complementary to the copper-shuttle hypothesis: the same tight binding that allows controlled delivery also constrains indiscriminate redox cycling of the metal in transit