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and Bejerot, S

for a pollution-centric survey, note these as mechanistic parallels rather than environmental exposures (Ramundo et al., 2024)

This amplifies the ATF4/CHOP/ChaC1 cascade under ER stress, resulting in decreased intracellular GSH and cysteine, increased intracellular ROS, redox homeostasis disruption, secondary oxidative stress in cancer cells and selective ferroptosis of cancer cells (66,100)