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This unprecedented precision stems from the peptide's targeting of a unique vulnerability in senescent cell biology, where FOXO4 prevents p53 from executing its normal pro-apoptotic function

This can cause damage to cells and tissues, leading to chronic inflammation. The activation of the Nrf2 pathway by sulforaphane reduces oxidative stress in a clever variety of ways, including pumping out more glutathione to neutralise free radicals, thereby reducing inflammation
