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Abstract The ketogenic diet (KD), a high-fat, low-carbohydrate diet, causes profound metabolic adaptations that go beyond energy production and affect endocrine function and thyroid hormone regulation

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Conceivable additional targets within the spike glycoprotein include the accessible cysteine residues and disulfide bond at C391-C525 and the bridge at C379-C432 [87], as well as other reactive residues such as methionine, histidine, tyrosine, and tryptophan [88]

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