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doi: 10.1055/s-2006-961482 [DOI] [PubMed] [Google Scholar] 64

Thus, MyD88 and TLR4 interact not directly but with the participation of TIRAP triggering the formation of a signaling complex

received funding from DFG under Germanys Excellence Strategy EXC-2049 390688087, Collaborative Research Center ReTune TRR 295-424778381, Clinical Research Group KFO 5023 BeCAUSE-Y, project 2 EN343/16-1

Increased IOP induces an inflammatory response of the glial population in the retina with the primary objective to isolate and resolve neural tissue damage by providing growth factors and metabolites to damaged RGCs, however, the chronification of this glial inflammatory response can affect glial cell function, neuronal support and extracellular glutamate buffering capacity (Martin et al., 2002), making RGCs and their axons more vulnerable and ultimately more prone to death by apoptosis (Neufeld and Liu, 2003