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Geffroy G, Benyahia R, Frey S, Desquiret-Dumas V, Gueguen N, Bris C, Belal S, Inisan A et al (2018) The accumulation of assembly intermediates of the mitochondrial complex I matrix arm is reduced by limiting glucose uptake in a neuronal-like model of MELAS syndrome

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Despite the limited research available on the interplay between mitochondria and iron overload, their critical function in maintaining iron homeostasis and managing associated damage underscores their importance in the pathological processes of both congenital and acquired iron overload

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