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In Hashimotos thyroiditis (HT), major sources of oxidative stress include misfolded proteins, dysbiosis and molecular mimicry, self-tolerance defects linked to AIRE gene dysfunction, and irregularities in thyroid protein folding (da Silva et al

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[DOI] [PubMed] [Google Scholar] 24.Fyfe J.C., Madsen M., Hjrup P., Christensen E.I., Tanner S.M., de la Chapelle A., He Q., Moestrup S.K

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In addition to genetic disease-risks, the identification of genetic disease-modifiers associated with disease complications, severity or prognosis provides crucial insights into the biological processes implicated in disease progression