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However, emerging research indicates that c-Rel may serve as a potential therapeutic checkpoint in MDSCs, selectively promoting pro-tumoral gene expression while repressing anti-tumoral genes via a c-Rel enhanceosome complex [87]
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[11] The peptides effects on the nitric oxide system contribute to vascular protection in research models
Overexpression of GSTs may contribute to increase detoxification of anticancer drugs 68
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This stability is attributed in part to the presence of proline residues, which help protect the peptide from enzymatic breakdown and support its potential as a research chemical in studies involving the gastrointestinal tract and other tissues
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HMGB1 exacerbates renal tubulointerstitial fibrosis through facilitating M1 macrophage phenotype at the early stage of obstructive injury
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