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doi:10.1001/jama.2021.4788

In line with the concept of redox-dependent hormesis, moderate oxidative stress can induce the NRF2-vitagene pathway, which upregulates a network of cytoprotective genes, including heat shock proteins, heme oxygenase-1, and thioredoxin [96]

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In particular, inflammatory processes seem to be involved in these symptoms

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