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Chronic treatment with fluoxetine decreases cerebral metabolic responses to the 5-HT1A agonist 8-hydroxy-2(di-N-propylamino)tetralin and increases those to the 5-HT2A/2C agonist 1-(2,5-dimethoxy-4-iodophenyl)-2-aminopropane and to the dopaminergic agonist apomorphine

This systematic review provides the most up-to date and extensive summary of all non-surgical treatments for PD, including only high-quality randomised trials

The most promising guidelines for strain development would include the overproduction of the carnitine activation enzymes (CaiB and CaiC), together with the trimethylammonium compounds transporter (CaiT) and the down-regulation of the expression of the glyoxylate shunt

L-carnitine - not only for athletes

doi: 10.1046/j.1365-201x.1999.00613.x