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Proteomic identification and quantification of S-glutathionylation in mouse macrophages using resin-assisted enrichment and isobaric labeling

Taken together, these results revealed that vitamin B12 promoted myelin regeneration after TBI

Further research and translational efforts are required to elucidate their therapeutic mechanisms of action and to evaluate their effectiveness in treating the acute and chronic phases of ABI

SEM, Standard Error of the Mean

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B12 deficiency can impair this process and prevent the use of storage form of folate when needed, resulting in functional folate deficiency