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Besides the increase in melanin content, AL lesions are characterized by epidermal alterations such as proliferation/differentiation disbalance or mitochondrial dysfunction, dermal photoaging marks, and disorganization of the dermal extracellular matrix (ECM) [21,22,23]. In particular, AL lesions present an impairment of the cutaneous structure with a marked elongation of the dermalepidermal junction (DEJ), an accumulation of melanin in the epidermis, especially in the rete ridges [21]

Clin Infect Dis (2012) 2012) 54:e13273

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Examining the downstream mechanisms through which MC1R-cAMP signalling antagonises inflammatory cytokine production and studying how MT-1s anti-inflammatory effects in skin cells contribute to its photoprotective biology beyond melanin-dependent UV absorption