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This bispecific Ab showed enhanced anti-tumor activity, reduced risk of immunogenicity and lacked endogenous biotin interference compared to the SA-biotin system

[5] Administering 100,000 IU intramuscularly establishes a high-capacity depot that may correct deficiency more predictably than oral dosing in malabsorption syndromes and maintains adequate circulating vitamin D over two to three months, an approach often referred to as Stoss therapy in endocrinology

Its mechanism appears to involve the stimulation of fat breakdown (lipolysis) while avoiding direct interaction with blood glucose metabolism

Biological repair and adaptation rarely depend on a single signaling molecule, and many modern studies increasingly focus on network-level interactions rather than isolated mechanisms