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Additionally, activating the SIRT1/Nrf2/p62 pathway can promote the healing of diabetic foot ulcers, possibly mediated by autophagy-dependent ferroptosis ( Together, the diverse roles of SIRT1 across multiple disease types underscore its significance as a promising therapeutic target
doi: 10.1093/jnci/djh320
iFSP1 treatment results in the obvious toxicity of RSL3 in a panel of genetically engineered (FSP1-knockout) human cancer cell lines
For central nervous system infections caused by the CARD9 defect, a combination of surgical drainage and long-term antifungal treatment is required (Shen et al., 2020)
Rational management of iron-deficiency anaemia in inflammatory bowel disease
