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We hypothesize that there is variability in the cerebral response to ischemia that is mediated by polymorphisms in genes encoding proteins which participate in this response

In Wilson disease the ATP7B gene is mutated resulting in a malfunctioning copper transporter at the trans-Golgi network

305 Most side chain oxidation products are hydrophilic and may therefore be more disruptive to protein conformation when they occur at residues with higher side chain hydrophobicity

The compounds have different stability profiles and pH characteristics