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An additional experiment conducted during this study showed that by treating 3T3-L1 adipocytes with NAC (600 M), a methylglyoxal impairment in insulin signalling was relieved, as shown by decreased insulin-induced insulin receptor substrate-1 tyrosine phosphorylation and decreased activity of phosphatidylinositol 3-kinase (66)

The results are not directly comparable to systemic Thymosin Beta-4 or TB-500 administration Key distinction: TB-500 is a synthetic fragment designed to mimic aspects of Thymosin Beta-4

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Payahoo, L., Khajebishak, Y

In the setting of chronic inflammatory disease states (e.g., Crohns disease or juvenile chronic arthritis), these diseases can also disturb the physiological synergy of the GH/IGF-I axis