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Therefore, the lipidome profiles may be considered as a potential biomarker tool to predict the progression of preclinical to established RA disease and facilitate monitoring of disease activity and treatment outcomes (191)

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48 On the other hand, other patients have minimal expansion of the mutant clone and have a lower percentage of circulating GPI-deficient RBCs

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Overexpression of p66 Shc leads to endothelial dysfunction, characterized by increased ROS production, elevated levels of E-selectin, and enhanced leukocyte transmigration across the human umbilical vein endothelial cell (HUVEC) monolayer [37]