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doi: 10.1007/BF03347357

Effects of resistance exercise training on ADRD biomarkers For the last 2 decades, the discourse related to the underlying mechanism for development of AD specifically has largely focused on the amyloid hypothesis, attributing the transition from MCI to AD to the accelerated rate of A plaque deposition and subsequent neurofibrillary tangle formation (Hardy and Higgins, 1992)

[] The importance of increasing glucagon secretion induced by AAs is that glucagon promotes rapid conversion of AAs to glucose, therefore, achieve even more glucose to the tissues

Given that many tenocytes expressing the gene scleraxis (SCX) play critical roles during tendon healing (Best et al., 2021), the researchers conditionally deleted the IGF-1R in SCX-expressing tenocytes by using a tamoxifen-inducible Cre-recombinase system