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Glutathione S-transferases (GSTs) are the enzymes expressed in the cytosol, microsomes, and mitochondria and catalyze glutathione conjugation to electrophilic compounds, not only produced from exogenous xenobiotics by biotransformation but also arise from endogenous substances

I would always tell patients, and still do, you know, there is such a thing called the placebo effect, right

The GPx-1 gene is not the only gene deleted in these LOH events, and a DNA repair gene, hOGG1, which may also repair oxidized DNA, is often deleted together with GPx-1

We hypothesized that low glutathione redox status would be associated with elevated markers of oxidative protein and DNA damage, inflammation and mitochondrial superoxide production in two regions that have been reported to be abnormal in autism, cerebellum and BA22

The development and maintenance of endometriosis are usually accompanied by abnormal fluctuations in these hormones and their receptors

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