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Gain-of-function mutations in oncogenes (such as RAS superfamily) or positive regulators (such as NF-B inducing kinase) contribute to sustained activation of NF-B signaling and subsequent cell proliferation

10.1007/s40519-023-01581-0 299

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Selenium treatment in animal models noted above reduces hyperalgesia, oxidative stress levels, and apoptosis by modulating these channels [138]