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It is characterized by the release of small portions of Ca 2+ and ROS from the mitochondria [184], prompting a short-term depolarization of the IMM that activates the ETC and inhibits ROS production by the mitochondria [185]
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10.1186/s13046-019-1505-4 34 FuchsG.ShemaE.VestermanR.KotlerE.WolchinskyZ.WilderS.et al (2012)
The systemic release of reactive oxygen species (ROS), proinflammatory cytokines, and other mediators amplifies multi-organ injury, leading to increased morbidity and mortality in various clinical contexts, including surgery, trauma, and vascular diseases [5,6]
Regulation of gamma-glutamyl-cysteine synthetase by nonallosteric feedback inhibition by glutathione
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