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at low physiological concentrations, zinc serves a twofold function of both neutralizing the negative charge density at the C terminal of -synuclein and assists other chaperones which work in a concerted manner to prevent aggregation of -synuclein, whereas at higher concentrations, zinc promotes fibrillation by binding to His50 at the N terminus and lowering its pK a , much like that observed in case of iron (Valiente-Gabioud et al., 2012)

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5.830.42%, P=0.0013) and had less cytoplasmic fragmentation after parthenogenetic activation (non-fragmentation rate 59.000.58% vs

Special consideration should be given to pre-existing renal disease [44] or an injured solitary kidney [45]

Measuring mucosal healing, cytokine levels, and tissue histology in combination protocols would clarify potential interactions