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Available data suggest that the targeting of the nicotinic-acetylcholine-receptor-mediated pathway may be a promising strategy for the prevention and treatment of CIPN-induced symptoms, in particular mechanical allodynia and intraepidermal nerve fiber loss [150]

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Other peptides act on the growth hormone and IGF-1 pathway, which is the real driver of hypertrophy in healthy adults

Restoring glutathione availability may lower oxidative burden, but clinical translation depends on concurrent micronutrient repletion (selenium, zinc, iron) and resolution of the primary inflammatory driver