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When intake and interference are accounted for and the elevation persists, the result is interpreted by weighing hepatic, renal, hematologic, malignant, and functional possibilities

10.1002/1873-3468.13123 43 HanY.TangX.ZhangY.HuX.RenL

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110 Despite a number of studies that reported somewhat conflicting results regarding the different cardiac phenotypes in response to increased AKT activity, it has currently been recognized that short-term appropriate AKT activation leads to physiological cardiac hypertrophy, while long-term AKT activation or AKT overexpression causes pathological cardiac hypertrophy, interstitial fibrosis, and even heart failure

495 As the role of GSK-3 in promoting A production and tau phosphorylation in AD models is recognized, selective inhibition of GSK-3 has emerged as a promising therapeutic strategy

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