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One model induces both mutant p53 and MDM2 haploinsufficiency during early development, highlighting MDM2's critical role in hematopoiesis, while the second model mimics CH by introducing mutant p53 in adult hematopoietic stem cells (HSCs)

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While there are some indications that it could have properties that might benefit heart health, the lack of targeted research means we cannot definitively say it is good for heart health
