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Lai, et al
10.1021/cr030441b Chem
The cell-level mechanism is now confirmed by two independent labs
10.1016/j.clindermatol.2019.04.007 40 FrancoA
These recurrent reductions in cerebral blood flow velocity can then precipitate ischemic changes, particularly in vulnerable border-zone areas and terminal arterial territories, especially in patients with a compromised hemodynamic reserve (e.g., those with intracranial arterial stenosis) [13]
