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observed a marked increase in ACSL4 expression in a heart failure model induced by TAC in mice, and ACSL4 overexpression in cardiomyocytes exacerbates the cardiac dysfunction triggered by pressure overload through ferroptosis

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In addition, cell viability, the expression of cell surface activation markers and cell proliferation were comparable in both WT and Gabrb3 cKO T cells after activation in vitro (Extended Data Fig

READ THE STUDY Elsevier | Journal of Clinical Orthopaedics and Trauma Study Year | 2020 | Thiago Se et al

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