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Mechanistic studies link disrupted one-carbon metabolism, elevated homocysteine, and mitochondrial dysfunction in B12 deficiency to pathways implicated in Alzheimers disease, supporting investigation of B12 in neurodegeneration [3]

DOI: Article Snippet: Patient derived T cells were isolated from B-ALL patients and cultured in lymphocyte medium KBM581 (Corning, USA) supplemented with 10% FBS and 50 IU/mL human interleukin-2 (IL-2) (R&D systems, USA). Qiao, Y., Chen, J., Wang, X., Yan, S., Tan, J., Xia, B., Chen, Y., Lin, K., Zou, F., Liu, B., He, X., Zhang, Y., Zhang, X., Zhang, H., Wu, X., & Lu, L

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HCV29RP and H101RP cells had an increased oxygen consumption rate (OCR) at baseline in comparison with parental cells

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