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Only one patient had a high enough elevation for a short-chain acyl-CoA dehydrogenase defect to be considered

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Loss of Drp1 leads to functional mitochondrial dysfunction and the accumulation of damaged fragmented mitochondria, caused by a weakened mitophagy (81)

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In parallel, GHK-Cu peptide apparently suppressed p38 MAPK phosphorylation, which is a pathway activated by inflammatory and oxidative stimuli and linked to cytokine production
