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The shift to MASLD aims to provide greater clarity in disease characterization, distinguishing it from other causes of liver fat accumulation while emphasizing its strong association with metabolic comorbidities, such as obesity, insulin resistance, dyslipidemia, and T2DM

found that SIRT1 ubiquitination reduces its expression during lipid metabolism in the liver (75)

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Our findings also revealed that exogenous L-serine supplementation significantly increased the survival rate of mice and decreased the colonization of P