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Stresul oxidativ contribuie la mbtrnirea prematur a esuturilor prin pierderea rezistenei i elasticitii acestora, la scderea activitii enzimatice, ct i n dezvoltarea diverselor forme de cancer (cauzat de mutaiile produse de radicalii liberi la nivelul ADN-ului celular, care determin activarea genelor oncogene i inactivarea genei oncosupresoare p53 [1]), n dezvoltarea bolilor cardiovasculare, bolilor inflamatorii, artritei, a fibrozei pulmonare sau a bolilor neurodegenerative precum Alzheimer i Parkinson

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These findings not only reveal distinct mechanisms of GSH in inhibiting cuproptosis and ferroptosis, but also suggest a potential combination strategy to suppress PDAC tumor growth.

This particular molecule has been synthesized to stop or limit the action of the NNMT enzyme (N-methyltransferase), which is involved in a series of biochemical reactions in the body, one of its main functions related to the metabolism of nicotinamide (a form of vitamin B3)