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gasseri pretreatment significantly inhibited RSL3-induced cell death (Fig

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Fat tissue is under constant pressure to maintain fluid balance or swelling will result

FIGURE 3 A newly identified GPX4-independent mitochondria-localized ferroptosis defense system, the DHODHCoQH2 system can compensate for GPX4 loss and detoxify mitochondrial lipid peroxides (Mao et al., 2021)

Endosomal maturation in primary inflammatory cells requires calcium-dependent binding of the sensor Munc13-4 to the late endosomal SNARE syntaxin 7 (STX7) 16 , a fusion modulator, and their binding is essential for endosomal signaling 16
