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Of note, a significant effect modification by sex was found between MEHOP exposure and the risk of thyroid malignancy ( p = 0.01) and for MEP in relation to the occurrence of thyroid benign nodules ( p = 0.03), with males having a higher risk than females in both conditions, probably due to the disruption of sex hormones by the two phthalate metabolites, although the small number of males requires caution in interpreting this result [114]

He/she can serve as a host in which the virus mutates making it more virulent, which when you're exposed a second time, may make you sick

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I am interested in finding the genes that determine these choices and their pattern in the margin, through overexpression changes in phenotype