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Patients with advanced prostate cancer are often treated with endocrine therapy including maximal androgen blockade (anti-androgens combined with castration) and classical androgen deprivation (orchiectomy or luteinizing hormone-releasing hormone agonists) [5]

Macrophage-specific p65 knockout (p65 f/f LysM-CreER T2 ) mice on the C57BL/6 background were generated by crossing floxed-p65 littermate (p65 f/f ) mice with LysM-cre mice

We have shown the difference between VRK2-WT cells and VRK2-KO cells in the sensitivity to GSH inhibition and noted that sensitive cells exhibited surged ROS levels following GSH inhibition (Fig