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At present, there is no definitive evidence of the interaction between the circadian clock system and the GH/IGF-1 axis and the pathophysiological mechanism causing metabolic disorders

While R273H mutation is non-functional, the P309S mutation is well tolerated [62]

Unlike the relatively stable hormonal environment in men, women experience cyclical variations in 17-estradiol (E2) and progesterone (P4) that directly influence key metabolic processes, including glucose homeostasis, lipid oxidation, amino acid turnover, and mitochondrial bioenergetics [3,4,5]

The immune response is localized to the proximal small intestine and causes the characteristic histologic findings of villous atrophy, crypt hyperplasia, and intraepithelial lymphocytosis

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