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The transcriptional activity of NF-kB p65 is inhibited by GYY4137 through s-sulfhydration at the cysteine 38 site

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While current treatments may alleviate specific physical or cognitive symptoms, they fail to halt disease progression, and curative solutions remain elusive

BT12 cells were treated with 1 M cisplatin and BT16 cells were treated with 10 M cisplatin due to their differential sensitivity to cisplatin and to induce a similar apoptotic rate

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