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In cultured VSMCs from rat aortas, GA treatment inhibited oxidative stress caused by hydroxyl radicals and promoted apoptosis, characterized by cytoplasmic shrinkage, vesicle formation, and nuclear condensation, thereby inhibiting the proliferation of vascular smooth muscle cells (Qiu et al., 2000)

Hollenbeck CB, Johnston P, Varasteh BB, Chen YD, Reaven GM

Furthermore, Rhodamine B-LNPs using rhodamine-B instead of BUD was prepared as red fluorescence for visualization

It is indicated that the two inhibitors might be promising probes for the biological and pharmacological roles of GSTM in cellular activity and as sensitizers for cisplatin-resistant ovarian cancer cells in chemotherapeutics, apart from the underlying mechanisms, are to be clarified

doi: 10.1016/j.cbi.2018.08.023

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