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Furthermore, the epidermis is a formative barrier that functions in preventing diffusion of molecules to the underlying dermis [2, 3]

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(1988), published in FEBS Letters , which found that GHK-Cu exposure at nanomolar concentrations in primary fibroblast cultures produced measurable increases in collagen synthesis relative to untreated controls.[5] This established the peptide's basic identity as a fibroblast-activating signal and prompted the wave of ECM-focused research that followed