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Ferroptosis signaling promotes functional recovery after sciatic nerve injury in mice Although axonal fusion after injury has not been directly demonstrated in vertebrates to date, there is extensive evidence showing that axonal fusion-like mechanisms can be induced to promote nerve repair in mammals, including humans 1

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In contrast, M2 macrophages produce cytokine signals like IL-6 and IL-10 that help cancer cells evade immune surveillance, recruit endothelial cells for angiogenesis, and avoid apoptosis, thus allowing them to proliferate more freely [146, 153]

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Other mechanisms of SIC In addition to the aforementioned mechanisms, calcium dysregulation, autonomic nervous system dysfunction and coronary microvascular disorders may be involved in SIC (54)