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The prooxidant activity of GGT has also recently been shown to promote the iron-dependent oxidative damage of DNA in GGT-transfected melanoma cells, thus potentially contributing to genomic instability and an increased mutation risk in cancer cells [50]

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[DOI] [PMC free article] [PubMed] [Google Scholar] 82.Cui S., Li W., Wang P., Lv X., Gao Y., Huang G

Consequently, it can be hypothesised that the administration of NAD + -boosting drugs may result in the regulation of ChREBP by means of pathways other than those involving the NAD +/Sirtuin pathways