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Various external environmental factors-including cigarette smoking, air pollution, and occupational dust exposure-can increase iron deposition within the respiratory tract, thereby elevating local iron load and promoting or exacerbating inflammatory responses (Zhang et al., 2019c)

Sikiric et al., 2018), along with the evidence that pentadecapeptide BPC 157 may prevent and/or attenuate or eliminate, thus, counteract both developing and already formed both arterial thrombosis (Hrelec et al., 2009

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For BPC 157-treated rats, in early and in late periods, the level of oxidative stress in the liver (MDA- and NO-levels, otherwise known to be increased and critical mediators of liver fibrosis[25]) was almost continuously within normal values as it was in rats with bile duct occlusion[25]

Thus, TMAO aggravates the formation of atherosclerotic plaques by aggravating the inflammatory immune response of macrophages and their transformation to foam cells