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In summary, these results indicate that GSH biosynthesis is essential for FH-deficient cells to overcome the oxidative stress caused by GSH succination

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10.1038/sj.leu.2402238 [DOI] [PubMed] [Google Scholar] Griffith O.W., and Mulcahy R.T

Some studies have suggested association between genetic polymorphism for GSTM1, GSTT1 and GSTP1 and DN, but others do not

This is precisely what one might expect to see if GPX1 is a protease substrate , because its cleavage would produce two fragments that would necessarily have reduced affinity to the enzyme relative to GPX1